Found this study that just made its way to pubmed.com It's consistent
with the points I've made on this NG time and time again.

"Nutrition. 2005 Feb;21(2):240-8. Related Articles,Links

Changes in lipid metabolism and antioxidant defense status in
spontaneously hypertensive rats and Wistar rats fed a diet enriched
with fructose and saturated fatty acids.

Girard A, Madani S, El Boustani ES, Belleville J, Prost J.

Universite de Bourgogne, UPRES Lipides Nutrition EA 2422, Faculte des
Sciences Gabriel, Dijon, France.

OBJECTIVE: Larger doses of fructose and saturated fat have been
associated with oxidative stress and development of hypertension. The
effects of modest amounts of fructose and saturated fatty acids on
oxidative stress are unknown. METHODS: To increase knowledge on this
question, 10-wk-old spontaneously hypertensive rats and Wistar rats
were fed for 8 wk with a control diet or an experimental diet enriched
with fructose (18%) and saturated fatty acids (11%; FS diet). The total
antioxidant status of organs and red blood cells was assayed by
monitoring the rate of free radical-induced red blood cell hemolysis.
Sensitivity of very low-density lipoprotein and low-density lipoprotein
(VLDL-LDL) to copper-induced lipid peroxidation was determined as the
production of thiobarbituric acid-reactive substances. Antioxidant
enzymes and vitamins were also measured to establish the oxidative
stress effect. RESULTS: The FS diet did not affect blood pressure in
either strain, but it increased plasma insulin concentrations only in
Wistar rats without affecting those of glucose of either strain. The FS
diet significantly enhanced plasma and VLDL-LDL triacylglycerol
concentrations without affecting concentrations of VLDL-LDL
thiobarbituric acid-reactive substances. The decreased content of
arachidonic acid and total polyunsaturated fatty acids in VLDL-LDL by
the FS diet may have prevented lipid peroxidation in this fraction.
Moreover, FS consumption by both strains was accompanied by a
significant increase in total antioxidant capacity of adipose tissue,
muscle, heart, and liver. This may have resulted from increased tissue
ascorbic acid levels and glutathione peroxidase and glutathione
reductase activities in tissues. CONCLUSIONS: These findings clearly
indicate that the FS diet did not alter blood pressure of spontaneously
hypertensive rats and Wistar rats. The FS diet resulted in
hypertriglyceridemia but increased the total antioxidant status, which
may prevent lipid peroxidation in these rats."