mdginzo wrote:

>>You realize I've sold over a million books. Do you really think I've
>
> spoken to that many people?
>
> It doesn't matter how many you have "spoken" to. The fact remains that
> you wouldn't sell any books if people didn't believe the crap you and
> your cult spew about low-carb diets.
>

Oh, and you might like to see the entire two articles that came up on
pubmed in response to "Atkins diet kidney":


A low-iron-available, polyphenol-enriched, carbohydrate-restricted diet
to slow progression of diabetic nephropathy.

Facchini FS, Saylor KL.

Department of Medicine, Division of Nephrology, San Francisco General
Hospital and University of California-San Francisco, Box 1342 UCSF, San
Francisco, CA 94143-1341, USA.

Diabetic nephropathy has become the leading cause of uremia. Several
lines of evidence suggest dietary factors other than protein intake have
a substantial role in the progression of diabetic nephropathy to
end-stage renal disease. The present investigation was initiated to
evaluate whether a carbohydrate-restricted, low-iron-available,
polyphenol-enriched (CR-LIPE) diet may delay and improve the outcome of
diabetic nephropathy to a greater extent than standard protein
restriction. To this aim, 191 diabetic patients, all with type 2
diabetes, were randomized to either CR-LIPE or standard protein
restriction and the following outcomes monitored: doubling of serum
creatinine, cumulative incidence of end-stage renal disease, and all
cause mortality. Over a mean follow-up interval of 3.9 +/- 1.8 years,
serum creatinine concentration doubled in 19 patients on CR-LIPE (21%)
and in 31 control subjects (39%) (P < 0.01). Renal replacement therapy
or death occurred in 18 patients on CR-LIPE (20%) and in 31 control
subjects (39%) (P < 0.01). These differences were independent from
follow-up interval, sex, mean arterial blood pressure, HbA(1c), initial
renal dysfunction, and angiotensin system inhibitor use. In conclusion,
CR-LIPE was 40-50% more effective than standard protein restriction in
improving renal and overall survival rates.


Effects of carbohydrate restriction on renal injury in the obese Zucker rat.

Kasiske BL, Cleary MP, O'Donnell MP, Keane WF.

The obese Zucker rat model of nonimmune-mediated, spontaneous focal
glomerulosclerosis is ideally suited to study the influence of diet on
the initiation and progression of glomerular injury. Young (6 wk) and
old (33 wk) lean and obese female Zucker rats were fed a
carbohydrate-restricted diet intermittently for 27 wk. Carbohydrate
restriction resulted in lower body weight (460 +/- 16 versus 310 +/- 7
g, p less than 0.025), kidney weight (1.26 +/- 0.04 versus 1.07 +/- 0.05
g, p less than 0.025), and glomerular area (6930 +/- 290 versus 5780 +/-
230 micron2, p less than 0.025) in young obese Zucker rats compared to
ad libitum-fed rats. Although urine-albumin excretion was substantially
reduced by carbohydrate restriction in young obese Zucker rats (41.1 +/-
12.3 versus 6.9 +/- 2.9 mg/24 h, p less than 0.01), glomerular injury
was not significantly altered. In old obese rats, carbohydrate
restriction did not significantly reduce albuminuria or prevent the
progression of glomerular injury. Thus, intermittent carbohydrate
restriction failed to alter significantly either the initiation of
glomerular injury in young, or the progression of nephron damage in old,
obese Zucker rats.

*******************

So while your doctor blames your kidney failure on your low carb diet,
there's not a lot of research to back him/her up. Certainly it has been
my experience that no matter what the health problem, from a cold to
residual pain from my car wreck, some doctor somewhere will see fit to
blame it on my low carb diet.

And you still haven't answered: Did you read the book and follow Atkins
as written, adding back progressively greater quantities of healthy
carbs every week, until you were in a very mild state of ketosis? Or
did you just decide that Induction was the whole diet, and call it "Atkins?"

Dana