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New York Times
January 5, 2004 Mad Cow Forces Beef Industry to Change Course By MICHAEL MOSS, RICHARD A. OPPEL JR. and SIMON ROMERO Jeffrey Behling, a dairy farmer in Washington State, used to burn the carcasses of his hobbled "downer" cattle until he found there was a market for their meat. Even so, selling damaged cows for human consumption never sat well with Mr. Behling, who in 2001 briefly had in his feedlot the Holstein cow identified last month as the downer with mad cow disease. "It's an absurd practice," Mr. Behling, 44, said in an interview. "Foolishness caused by maybe a certain amount of greed." The financial motive that drove the industry to defend practices like selling downers has been turned on its head by the discovery of mad cow disease. Now, in an attempt to rescue the market for American beef, the industry is being forced to accept regulation it has long fought. But some large American companies that process and sell beef had already abandoned those more controversial practices, which had been a rallying point for food safety advocates since mad cow disease appeared overseas nearly two decades ago. While a schism developed in the industry, the current crisis reveals how government regulators sided with companies that adhered to those methods of operation. When an animal rights group, Farm Sanctuary, and an individual, Michael Baur, sued the government to force a ban on using downer animals for food, government lawyers persuaded a federal judge to dismiss the case on the ground that mad cow disease, or bovine spongiform encephalopathy, had not appeared in the United States. "The threat of B.S.E. from downed livestock is not `real and immediate,' " the lawyers argued. "B.S.E. has never been found in the country's livestock, and there is no reasoned basis to expect that it ever will be considering the measures being taken against it." An appeals court reinstated the case on Dec. 16, 2003 - one week before the announcement that the disease had been discovered. For years, the industry had a simple strategy: Fight proposals that would crimp its ability to squeeze as much revenue as possible from each cow. The finances were compelling. At least 150,000 downer cattle - those who because of injury or illness cannot walk - were sold annually for human consumption for as much as a few hundred dollars apiece, extra money for cattlemen struggling with low prices. Food safety advocates warned that these cattle could carry disease, but the political power of the industry was evident in 2002 when its lobbyists helped defeat legislation banning the commercial slaughter of downer cattle even after it had been approved by the House and the Senate. In the 1990's, meatpackers bought machines that were able to strip a few extra pounds off carcasses while saving millions in labor costs. Critics tried to limit the use of the so-called advanced meat recovery systems, citing studies showing that the extra meat was sometimes laced with nerve tissues, where mad cow disease can incubate. But by one consultant's account several years ago, getting rid of the machines would mean a loss to the industry of more than $130 million a year. Now the money saved by fighting those changes is dwarfed by the billions the industry stands to lose unless it can convince consumers, especially overseas, that its beef is safe. "They played a high-risk, high-stakes game, and they lost their bet," said Representative Gary L. Ackerman, a New York Democrat who pushed for a ban on the commercial slaughter of downer cows. "Now the perception among millions of people is that this product isn't safe, and they can't put Humpty Dumpty back together again." It Was the Best of Times As part of the campaign to restore consumer confidence, Agriculture Secretary Ann M. Veneman last week banned the use of downer cattle for meat and imposed further regulation on advanced recovery systems. Still, after the disease was detected last month, cattle prices plunged about 20 percent, while the $3.6 billion export market for beef, veal and variety meats largely evaporated, according to Cattle-Fax, an industry research firm. This came after United States beef prices had reached record highs, partly because of the restriction of imports from Canada after the mad cow outbreak there and the rising popularity of beef-friendly eating trends like the Atkins diet. "The last year had been heaven on earth for beef producers," said Don Stull, a co-author of "Slaughterhouse Blues," a study of the meat industry. But even in the best of times, meatpacking remains a cutthroat business. Steve Kay, the publisher of Cattle Buyers Weekly, estimates that profit margins rarely climb above 2 percent as companies deal with fluctuating cattle prices and relatively higher labor costs. Those financial constraints, which led meatpackers to harvest every last pound of meat, also caused consolidation in the industry. Five meatpackers now slaughter more than 80 percent of the nation's steers and heifers: Tyson, Excel, Swift, National Beef Packing and Smithfield. Bigger slaughterhouses have cut processing costs by as much as 40 percent, according to Agriculture Department data. Wholesale beef prices have declined almost every year since the early 1980's. "We have the cheapest food supply in the world in terms of what we spend on food as part of our incomes," said Dean Cliver, a professor of population health at the University of California at Davis. Affordable beef has helped make for easy relations between the industry and federal regulators. According to the Center for Science in the Public Interest, a consumer group, a dozen top officials of the Department of Agriculture have worked or lobbied for the industry or for industry trade groups. They include Jim Moseley, the deputy agriculture secretary, who was managing director of Infinity Pork LLC, a hog farm; Dr. Chuck Lambert, the deputy under secretary for marketing and regulatory programs, who was chief economist of the National Cattlemen's Beef Association; and Mary Waters, the assistant secretary for Congressional relations, who was senior director and legislative counsel for ConAgra Foods. "It's not surprising the industry has so much influence given the number of U.S.D.A. officials who have been hired directly out of the meat industry," said Caroline Smith DeWaal, the center's food safety director. Alisa Harrison, the department's press secretary, said Secretary Veneman set policy by consulting a wide range of advisers and interest groups. "To make a sweeping charge that her decisions are influenced just because she has people from industry on her staff is very disingenuous," she said. She also noted that the department's top food safety official, Dr. Elsa A. Murano, had been director of the Center for Food Safety at Texas A&M University. Ms. Harrison also said the department had been attentive to the dangers of mad cow well before last month. "We were able to make the quick announcement that we did last week because a lot of the groundwork had been going on" since the discovery in May of a cow in Canada with the disease, she said. "These are things we have been looking at." But the debate over the advanced recovery system shows how the industry and regulators have resisted pressure from safety advocates since the disease appeared in Britain in 1986 and then spread to 18 other European countries. New Process, New Concerns The technology, developed a decade ago, uses hydraulic pressure to force extra pounds off cow carcasses, producing filler for processed foods like hamburger, hot dogs and pizza toppings. Consumer groups initially complained that bone was getting into the advanced meat recovery product and argued that the product should not be labeled as beef. Then, in 1997, federal agriculture officials announced that they had found spinal cord tissue in some of the meat. Concerned that the nerve tissue could increase the public's risk of contracting mad cow disease, consumer groups asked the government to ban the technology, said Linda Golodner, president of the National Consumers League. But both the industry and government regulators resisted, arguing that the absence of the disease in the United States showed that there was no problem. "For us, so far, it's a non-public-health issue because we have no B.S.E.," Kaye Wachsmuth, who was then deputy administrator for public health science at the Agriculture Department, said in 1998. There were other arguments against the ban. The machinery replaced workers who could suffer crippling injury from trimming the carcasses by hand; one consultant study estimated that 394 workers would be injured if slaughterhouses returned to hand-trimming. Companies that sell the machines say such beef poses no threat. "The accepted science essentially states that there is not any relationship between B.S.E. and A.M.R.," said Harold T. Hodges, vice president of government relations and product quality for the BFD Corporation, one of the distributors of the machines. "We've never had an issue." Proponents of the technology argued that proper enforcement of the technology, rather than a ban, could prevent contamination. "It's always been a legitimate enforcement compliance issue to ensure that what you call beef is beef," said Robert Hibbert, a lawyer who represented meat processors that used the technology. "There is no justification for banning something on the basis that it has been removed by a machine rather than by hand with a knife." But some industry officials worried that not every processor used the machinery properly. At an American Meat Institute conference in Chicago in 1997, an executive of a major beef producer warned that applying too much pressure would force bone material into the beefy mush. In addition, the spinal cord has to be carefully removed before the cow carcass is fed to the machine. Second Thoughts As federal officials continued to find traces of nervous-system tissue in recovered beef, some companies determined that the potential cost of these practices outweighed the gains. With consumer groups pressing for a boycott of meat produced using advanced recovery technology, a host of restaurants and producers announced they were advanced meat recovery free, including General Mills and McDonald's, which swore off downer-cow meat as well. In a fact sheet, McDonald's says, "These policies meet or exceed all government requirements, and have been reviewed by our international scientific council on B.S.E., made up of renowned experts in this field." Meanwhile, some slaughterhouses had other reasons to stop using the machines. In late 2002, Shapiro Packing, a processor in Augusta, Ga., produced tainted beef using the machinery system. The contaminated material was destroyed, but the company had to spend a lot of money to shore up its operation, said Dane Bernard, vice president for food safety at Keystone Foods, which manages Shapiro Packing. Additional workers were placed on the line to ensure that the carcasses were properly stripped of their spinal cords, and the company's inspections became nearly continuous, Mr. Bernard said. The new measures increased expenses while big beef buyers were boasting that their food was not processed using advanced meat-recovery systems. So last summer, Shapiro mothballed its machinery and returned to manual trimming. "I can't say we had a crystal ball," Mr. Bernard said. "Sometimes it's better to be lucky than good." The discovery of mad cow disease is likely to increase the debate over the technology. Dr. Wachsmuth, the agriculture official who defended the technology in 1998, said in an interview on Saturday that the absence of the disease had been an important factor in that defense. "The mere threat of it wasn't enough," said Dr. Wachsmuth, who is now retired. "Now that we do have B.S.E., maybe it should be revisited." Dan Murphy, a spokesman for the American Meat Institute, the meatpackers' trade group, said the number of processors using the technology had recently fallen to fewer than 30 from 35. He said that the machines once produced several hundred million pounds of meat a year, but that a survey in late 2002 found the number had dropped to 45 million. Even so, he said, "We're confident that this is a safe, wholesome product that doesn't trigger any concern or carry any danger in its use." But he acknowledged that some members of the association were less supportive: "There are companies that would just as soon we said nothing." In her announcement last week, Secretary Veneman imposed regulations intended to further keep unwanted tissue from the food supply, but she stopped short of a ban on the technology. Mr. Murphy, the industry spokesman, acknowledged that a further review of the technology was possible, especially if there is pressure from overseas trading partners. "Nobody is going to give up $1.2 billion in beef trade for a handful of A.M.R.," he said. In Washington State, Mr. Behling, the onetime holder of the diseased cow, said that in the days since the discovery of mad cow, the industry has learned that lesson in global economics. Mr. Behling, who has a few thousand cows in his operation, said that when the occasional downer cow appeared, a slaughterer would drive out to his farm with a hoist and give him $100 for the hobbled animal. But in the wake of the mad cow crisis, he said, "My feeling is that any money that dairy farmers might have made from downer cows, they gave it all back this week." ================================================== ====== |
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Is it still only one cow that has been found in the United States to have
the disease? I mean, the article (what of it I read, it was very boring) made it sound as if the was some actual danger. Maybe thousands were found to be infected and I didn't read about it yet? |
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![]() "Bill" > wrote in message t... > Is it still only one cow that has been found in the United States to have > the disease? I mean, the article (what of it I read, it was very boring) > made it sound as if the was some actual danger. Maybe thousands were found > to be infected and I didn't read about it yet? There's been speculation that many of the people who were diagnosed with Alzheimer's in the US may have actually had CJD. I thought I read something about the brains of people who had died from Alzheimer's being studied and the ones doing it finding out they had died of CJD, but I can't remember where the article is now. -Rubystars |
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![]() "Rubystars" > wrote in message y.com... > > "Bill" > wrote in message > t... > > Is it still only one cow that has been found in the United States to have > > the disease? I mean, the article (what of it I read, it was very boring) > > made it sound as if the was some actual danger. Maybe thousands were > found > > to be infected and I didn't read about it yet? > > There's been speculation that many of the people who were diagnosed with > Alzheimer's in the US may have actually had CJD. I thought I read something > about the brains of people who had died from Alzheimer's being studied and > the ones doing it finding out they had died of CJD, but I can't remember > where the article is now. > > -Rubystars > There is no proof what so ever that those inflicted with Alzheimer's had in fact CJD. There was one cow found to be diseased. They said millions would die in England when they had their scare and they had thousands of diseased cattle. They had a couple of deaths there. More liberal media hocus pocus. We had one cow in Canada and not one illness. These **** wads in the media don't care how they sell papers or get ratings. Jeff |
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Jeff > writes
>There is no proof what so ever that those inflicted with Alzheimer's had in >fact CJD. It's not unknown, apparently, for people who have died of alzheimers to also be showing some CJD-like pathology. They probably have heart disease too .... >There was one cow found to be diseased. They said millions would >die in England when they had their scare Some people said 10's of millions (a vegetarian professor of microbiology for example). >and they had thousands of diseased >cattle. Actually about 170,000 clinical cases of BSE, probably about a million counting sub-clinical cases. >They had a couple of deaths there. Under 150 over 10 years, about 20/year in a population of 60M. >More liberal media hocus pocus. >We had one cow in Canada and not one illness. These **** wads in the media >don't care how they sell papers or get ratings. This is normal. It so easy to sell copy by producing scare stories written by ignorant journalists for an ignorant and fearful population that they seem to find it irresistible. I do hope someone sues a few newspapers, there would be a cheer from across the water. -- Oz This post is worth absolutely nothing and is probably fallacious. DEMON address no longer in use. |
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On Mon, 5 Jan 2004 03:00:20 -0600, "Jeff" > wrote:
> >"Rubystars" > wrote in message gy.com... >> There's been speculation that many of the people who were diagnosed with >> Alzheimer's in the US may have actually had CJD. I thought I read >>something >> about the brains of people who had died from Alzheimer's being studied and >> the ones doing it finding out they had died of CJD, but I can't remember >> where the article is now. >There is no proof what so ever that those inflicted with Alzheimer's had in >fact CJD. We must be careful not to mix up things. There is a disease we can call CJD (classical CJD), and another distinct from it, called vCJD (variant CJD). Undoubtedly there have been people diagnosed with Alzheimer's who really died from CJD, but not likely any from vCJD. vCJD affects relatively young people. >There was one cow found to be diseased. They said millions would >die in England when they had their scare and they had thousands of diseased >cattle. <snip> Pointedly one could say some said there was only a relatively few BSE clinical cases at one stage during the UK epizootic , with the case number appearing to be leveling out at a low prevalence. Downplaying it, so maybe it was no big deal, one could wait and see. However, they were sitting on a huge number of subclinical cases, the proverbial hidden part of the ice-berg they just could not see yet, and we are to be happy that some were sufficiently foresighted to act accordingly. It is difficult to come to terms with a disease with long incubation, and more so, when the disease is new. When vCJD first emerged it was known that people in England had eaten 100s of thousands of subclinical BSE affected cattle. Necessarily any reasonably estimated ranges for the possible scope of the newly discovered disease would have to be wide. In either case complacency, leaning to the low end would have to be out of the question. |
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Steve Wertz wrote:
> Bice to see your MO hasn't changed. > > This idiots sole purpose in life is to post articles he clips off the > internet. He always uses a different name, never has any thoughts of > his own to add, and crossposts to wildly off-topic groups. > > The guy is nothing but troll. > > -sw Yeah, he's a vegetarian with an inferiority complex. Feels he has to masturbate at the keyboards for entertainment. BTW, he's a plagerist also. Betting he does not have the sources permission to cut and paste this. jim |
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OrionCA > writes
>The # of cases of human illness linked to BSE-infected meat is exactly >-0-, even in the UK where the largest number of "Mad Cows" were found. Thats simply untrue. They are linked, not absolutely proven but more than good enough evidence to base a strategy on. >There's about 10 cases Under 150 cases > of neurological disorders that they SUGGEST >could be linked to BSE but there's no evidence that these people ever >consumed the "tainted" brain tissue so it's all speculation. No, their prion was quite a good match for BSE, and the case curve shape is what you would expect if BSE was the cause. >Meanwhile there are thousands of cases of people getting sick each >year - and even dying - from consuming spoilt or tainted beef. People >are in more danger of being injured by an angry housewife dropping a >frozen rump roast on their heads from a 4th story apartment window >than being injured by meat from a so-called "Mad Cow". That's true, but remember the UK *DID* institute controls to reduce human exposure very considerably. So please try to keep rational. -- Oz This post is worth absolutely nothing and is probably fallacious. DEMON address no longer in use. |
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![]() "Torsten Brinch" > wrote in message ... > On Mon, 5 Jan 2004 03:00:20 -0600, "Jeff" > wrote: > > > > >"Rubystars" > wrote in message > gy.com... > > >> There's been speculation that many of the people who were diagnosed with > >> Alzheimer's in the US may have actually had CJD. I thought I read > >>something > >> about the brains of people who had died from Alzheimer's being studied and > >> the ones doing it finding out they had died of CJD, but I can't remember > >> where the article is now. > > >There is no proof what so ever that those inflicted with Alzheimer's had in > >fact CJD. > > We must be careful not to mix up things. There is a disease we can call > CJD (classical CJD), and another distinct from it, called vCJD (variant > CJD). Undoubtedly there have been people diagnosed with Alzheimer's > who really died from CJD, but not likely any from vCJD. vCJD affects > relatively young people. > > >There was one cow found to be diseased. They said millions would > >die in England when they had their scare and they had thousands of diseased > >cattle. <snip> > > Pointedly one could say some said there was only a relatively few BSE > clinical cases at one stage during the UK epizootic , with the case > number appearing to be leveling out at a low prevalence. Downplaying > it, so maybe it was no big deal, one could wait and see. However, they > were sitting on a huge number of subclinical cases, the proverbial > hidden part of the ice-berg they just could not see yet, and we are to > be happy that some were sufficiently foresighted to act accordingly. note that when people have their tonsils out, these are now, in many parts of the UK if not all of it, tested for nvCJD.. The idea being that these tissues are among the first parts to become infective and therefore will be a useful marker as to the number of cases. So far none have come up infected Jim Webster |
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C.W. saw Sally selling seashells by the seashore and told us all about
it on Mon, 05 Jan 2004 05:39:41 GMT: I'm not going to say anything about Mad Cows, because it's such a rare disease that I don't think we need to panic... but the idea of eating sick animals is revolting. It's enough to make me want to evict beef from my diet (I hate the taste and only eat it because my family insists that it's 'good for you' and that I need the iron...) It's just GROSS... ~Karen AKA Kajikit Nobody outstubborns a cat... Visit my webpage: http://www.kajikitscorner.com Allergyfree Eating Recipe Swap: http://groups.yahoo.com/group/Allergyfree_Eating Ample Aussies Mailing List: http://groups.yahoo.com/group/ampleaussies/ |
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![]() "JimLane" > wrote in message ... > Steve Wertz wrote: > > Bice to see your MO hasn't changed. > > > > This idiots sole purpose in life is to post articles he clips off the > > internet. He always uses a different name, never has any thoughts of > > his own to add, and crossposts to wildly off-topic groups. > > > > The guy is nothing but troll. > > > > -sw > > Yeah, he's a vegetarian with an inferiority complex. Feels he has to > masturbate at the keyboards for entertainment. BTW, he's a plagerist > also. Betting he does not have the sources permission to cut and paste > this. Just so you know he doesn't have to. Copyright laws permit "fair use" for educational purposes. Disseminating publications like that one fall under that category. Paul |
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![]() "OrionCA" > wrote in message ... > On Mon, 05 Jan 2004 06:36:23 GMT, "Bill" > > wrote: > > >Is it still only one cow that has been found in the United States to have > >the disease? I mean, the article (what of it I read, it was very boring) > >made it sound as if the was some actual danger. Maybe thousands were found > >to be infected and I didn't read about it yet? > > There are 3 herds (so far) that may have consumed feed with cattle > meat in it before the 1997 ban took effect. The "Mad Cow" came from > one of those herds. They're pretty sure the cow came from Canada > which had the same problem switching over to pure-vegetable based feed > after the UK "Mad Cow" scare. There's been no evidence that any other > cattle from these herds is infected but the USDA is still testing > them. All three herds have been quarantined and are scheduled to be > destroyed when the investigation is complete. > > The bad news is that meat from these herds has been found in > supermarkets across the Western United States. The good news is > there's no proof that "Mad Cow" disease can be caught by humans. > Nevertheless the authorities are being hyper-cautious about and > recalling all the potentially tainted meat they can find. Tell that to the 150 or so people who died from it in England in the 80s. It most definitely does infect humans. Paul |
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![]() "Oz" > wrote in message ... > Jeff > writes > > >There is no proof what so ever that those inflicted with Alzheimer's had in > >fact CJD. > > It's not unknown, apparently, for people who have died of alzheimers to > also be showing some CJD-like pathology. They probably have heart > disease too .... > > >There was one cow found to be diseased. They said millions would > >die in England when they had their scare > > Some people said 10's of millions (a vegetarian professor of > microbiology for example). > > >and they had thousands of diseased > >cattle. > > Actually about 170,000 clinical cases of BSE, > probably about a million counting sub-clinical cases. > > >They had a couple of deaths there. > > Under 150 over 10 years, about 20/year in a population of 60M. > > >More liberal media hocus pocus. > >We had one cow in Canada and not one illness. These **** wads in the media > >don't care how they sell papers or get ratings. So you're saying that if prions could vote, they'd vote democrat? This reminds me so much of the imbeciles that refuted the devastating affects of DDT back in the 60's. They were the one ultimately proven wrong, now weren't they? One stalwart individual even sprinkled it on his breakfast cereal to prove it was all just "hocus pocus." He died, at a very young age, of liver cancer a few years later. Basic husbandry practices in Europe dating back 8 centuries dictated that animals not be fed the remains of their own species. Seems a few lessons have been forgotten. But by all means label every scientist out to enlighten and protect the public some wild-eyed radical. Have some DDT with your Cheerios and think about it. Paul |
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![]() "Paul M. Cook©®" > wrote in message ... > > So you're saying that if prions could vote, they'd vote democrat? As Oz is a Brit, this comment is as meaningless as those I have snipped I suggest you actually check a few facts Jim Webster |
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Paul M. Cook©® > writes
> >So you're saying that if prions could vote, they'd vote democrat? Eh? >This >reminds me so much of the imbeciles that refuted the devastating affects of >DDT back in the 60's. They were the one ultimately proven wrong, now >weren't they? One stalwart individual even sprinkled it on his breakfast >cereal to prove it was all just "hocus pocus." He died, at a very young >age, of liver cancer a few years later. BSE and prions are not fresh news to the UK. Take a look at the date of the first case. >Basic husbandry practices in Europe dating back 8 centuries dictated that >animals not be fed the remains of their own species. Pigs have been fed pigswill containing pork for much longer than that. Chickens will happily cannibalise each other. So you are patently wrong. That's even before we get into the well documented cases of ruminants knawing ones and eating eggs and chicks. >Seems a few lessons >have been forgotten. Seems you are a tad short of actually knowing what you are talking about. >But by all means label every scientist out to >enlighten and protect the public some wild-eyed radical. Hardly. >Have some DDT with your Cheerios and think about it. Another effort at emotive nonsense. -- Oz This post is worth absolutely nothing and is probably fallacious. DEMON address no longer in use. |
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"Jeff" > wrote in message ...
> > "Rubystars" > wrote in message > y.com... > > > > "Bill" > wrote in message > > t... > > > Is it still only one cow that has been found in the United States to have > > > the disease? I mean, the article (what of it I read, it was very boring) > > > made it sound as if the was some actual danger. Maybe thousands were > > > found to be infected and I didn't read about it yet? > > > > There's been speculation that many of the people who were diagnosed with > > Alzheimer's in the US may have actually had CJD. I thought I read something > > about the brains of people who had died from Alzheimer's being studied and > > the ones doing it finding out they had died of CJD, but I can't remember > > where the article is now. > > > > -Rubystars > > > There is no proof what so ever that those inflicted with Alzheimer's had in > fact CJD. There was one cow found to be diseased. They said millions would > die in England when they had their scare and they had thousands of diseased > cattle. They had a couple of deaths there. More liberal media hocus pocus. > We had one cow in Canada and not one illness. These **** wads in the media > don't care how they sell papers or get ratings. > > Jeff Alzheimer's And CJD Scientifically Linked By Michael Greger 12-29-03 (First published 6-16-96) If indeed a form of Bovine Spongiform Encephalopathy (BSE) exists in the United States, one might expect to see a rise in the number of cases of Creutzfeldt-Jakob disease (CJD). CJD, however, is not a reportable illness in this country (Holman, 1995). Because the Centers for Disease Control (CDC) does not actively monitor the disease (Altman, 1996d) a rise similar to the one in Britain could be missed (Altman, 1996d). Already, a number of U.S. CJD clusters have been found. In the largest known U.S. outbreak of sporadic cases to date(Flannery, 1996) a five-fold expected rate was found to be associated with cheese consumption in Pennsylvania's Lehigh Valley (Little, 1993) A striking increase in CJD was also reported in Florida (Berger, 1994) and there is an anecdotal report of an cluster in Oregon (Boule, 1996). An analysis of death certificates in a number of states, though, showed an overall stable and typical CJD incidence rate from 1979 to 1993 (World, 1996). To track the disease, the CDC has just initiated a four-state study of death certificates (Altman, 1996a), but since it is considered well known that death-certificate diagnoses are not always accurate (Davanpour, 1993) the survey may not provide an accurate assessment. The true prevalence of prion diseases in this or any other country remains a mystery (Harrison, 1991). Compounding the uncertainty, autopsies are rarely performed on atypical dementias (Harrison, 1991), because medical professionals fear infection (Altman, 1996a). The officially reported rate in this country is less than 1 case in a million people per year (World, 1996). An informal survey of neuropathologists, however, registered a theoretical range of 2-12% of all dementias as actually CJD (Harrison, 1991). And hundreds of thousands of Americans suffer from severe dementias every year (Brayne, 1994; United, 1995). Two other studies average about a 3% CJD rate among dementia patients (Mahendra, 1987; Wade, 1987). A preliminary 1989 University of Pennsylvania study showed that 5% of patients diagnosed with dementia were actually dying from Creutzfeldt-Jakob disease (Boller, 1989). It would seem CJD is seriously underdiagnosed at present (Harrison, 1991). The most common misdiagnosis of CJD is Alzheimer's disease (Harrison, 1991). CJD was even described by our government's top CJD researcher (Wlazelek, 1990a) as "Alzheimer's in fast forward (Wlazelek, 1990b)." The symptoms and pathology of both diseases overlap (Brown, 1989). There can be spongy changes in Alzheimer's, for example, and senile plaques in CJD (Brown, 1989). The causes may overlap as well; epidemiological evidence suggests that people eating meat more than four times a week for a prolonged period have a three times higher chance of suffering a dementia than long-time vegetarians (Giem, 1993), although this result may be confounded by vascular factors (Van Duijn, 1996). Paul Brown, medical director for the U.S. Public Health Service (Gruzen, 1996), said that the brains of the young people who died from the new CJD variant in Britain even look like Alzheimer's brains (Hager, 1996). Stanley Prusinger, the scientist who coined the term prion, speculates Alzheimer's may in fact turn out to be a prion disease (Prusiner, 1984). In younger victims the disease could look like multiple sclerosis or a severe viral infection, according to Alzheimer's expert Gareth Roberts (Brain, 1996). An estimated two to three million Americans are afflicted by Alzheimer's (Scully, 1993); it is the fourth leading cause of death among the elderly in the U.S (Perry, 1995). Twenty percent or more of people clinically diagnosed with Alzheimer's disease are found at autopsy to not have had Alzheimer's at all (McKhann, 1984). At Yale, out of 46 patients clinically diagnosed with Alzheimer's, 6 were proven to be CJD at autopsy (Manuelidis, 1989). In another post-mortem study 3 out of 12 "Alzheimer" patients actually died from a spongiform encephalopathy (Teixeira, 1995). Carleton Gajdusek, who was awarded a Nobel Prize for his work with prion diseases (Manuelidis, 1985), estimates that 1% of people showing up in Alzheimer clinics actually have CJD (Folstein, 1983). That means that hundreds* of people (Hoyert, 1996; United, 1995) may already be dying from mad cow disease each year in the United States. WORKS CITED Altman, Lawrence K. "Four States Watching for Brain Disorder." New York Times, 9 April 1996a. Altman, Lawrence K. "U.S. Officials Confident That Mad Cow Disease of Britain Has Not Occured Here." New York Times, 27 March 1996d: 12A. "Apocalypse Cow: U.S. Denials Deepen Mad Cow Danger." PR Watch, 3.1 (1996): 1-8 Berger, Joseph R., et al. "Creutzfeldt-Jakob Disease: A Ten-Year Experience." Neurology, 44 (1994): A260. Bleifuss, Joel. "Killer Beef." In These Times, 31 May 1993: 12-15. Boller, F., O. L. Lopez, and J. Moossy. "Diagnosis of Dementia. " Neurology, 38 (1989): 76-79. Boule, Margie. "Despite Anectdotal Evidence, Docs Say No Mad Cow Disease Here." Oregonian, 16 April 1996: C01. "Brain Disease May Be Commoner Than Thought - Expert." Reuter Information Service, 15 May 1996. Brayne, C. "How Common are Cognitive Impairment and Dementia?" Dementia and Normal Aging, Canbridge: University Press, 1994. Brown, Paul. "Central Nervous System Amyloidoses." Neurology, 39 (1989): 1103-1104. Davanpour, Zoreth, et al. "Rate of Creutzfeldt-Jakob Disease in USA." Neurology, 43 (1993): A316. Flannery, Mary. "Twelve - Fifteen 'Mad Cow' Victims a Year in Area." Philadelphia Daily News, 26 March 1996: 03. Folstein, M. "The Cognitive Pattern of Familial Alzheimer's Disease." Biological Aspects of Alzheimer's Disease. Ed. R. Katzman. Cold Spring Harbor Laboratory, 1983. Gruzen, Tara. "Sheep Parts Fail to Cause Mad Cow Disease in U. S. Test." Seattle Times, 29 March 1996: A11. Hager, Mary and Mark Hosenball. "'Mad Cow Disease' in the U.S.?" Newsweek, 8 April 1996:58-59. Harrison, Paul J., and Gareth W. Roberts. "'Life, Jim, But Not as We Know It'? Transmissible Dementias and the Prion Protein." British Journal of Psychiatry, 158 (1991): 457-70. Holman, R. C., et al. "Edidemiology of Creutzfeldt-Jakob Disease in the United States, 1979-1990." Neuroepidemiology, 14 (1995): 174-181. Hoyert, Donna L. "Vital and Health Statistics. Mortality Trends for Alzheimer's Disease, 1979-1991." Washington: U.S. Dept. of Health and Human Services, 1996. Little, Brian W., et al. "The Epidemiology of Creutzfeldt-Jakob Disease in Eastern Pennsylvania." Neurology, 43 (1993): A316. Mahendra, B. Dementia, Lancaster: MTP Press Limited, 1987: 174. Manuelidis, Elias E. "Presidential Address." Journal of Neuropathology and Experimental Neurology, 44 (1985): 1-17. Manuelidis, Elias E. and Laura Manuelidis. "Suggested Links between Different Types of Dementias: Creutzfeldt-Jakob Disease, Alzheimer Disease, and Retroviral CNS Infections." Alzheimer Disease and Associated Disorders, 2 (1989): 100-109. McKhann, Guy., et al. "Clinical Diagnosis of Alzheimer's Disease." Neurology, 34 (1984): 939. Prusiner, S. "Some Speculations about Prions, Amyloid, and Alzheimer's Disease." New England Journal of Medicine, 310 (1984): 661-663. Perry, R.T., et al. "Human Prion Protein Gene: Two Different 24 BP Deletions in an Atypical Alzheimer's Disease Family." American Journal of Medical Genetics, 60 (1995): 12-18. Scully, R. E., et al. "Case Records of the Massachusetts General Hospital." New England Journal of Medicine, 29 April 1993: 1259-1263. Teixeira, F., et al. "Clinico-Pathological Correlation in Dementias." Journal of Psychiatry and Neuroscience, 20 (1995): 276-282. United States Department of Commerce. Statistical Abstract of the United States, Washington: Bureau of the Census, 1995. Van Duijn, C. M. "Epidemiology of the Dementia: Recent Developments and New Approaches." Neuroepidemiology, 60 (1996): 478-488. Van Duijn, C. M. "Epidemiology of the Dementia: Recent Developments and New Approaches." Neuroepidemiology, 60 (1996): 478-488. Wade, J. P. H., et al. "The Clinical Diagnosis of Alzheimer's Disease." Archives of Neurology, 44 (1987): 24-29. Wlazelek, Ann. "Fatal Brain Disease Mystifies Experts." Morning Call, 23 September 1990a: B01. Wlazelek, Ann. "Scientists Try to Track Fatal Disease; International Expert Visits Area to Study Unusual Incedence Rate." Morning Call, 27 September 1990b: B04. "World Health Organization Consultatoin on Public Health Issues Related to Bovine Spongiform Encephalopathy and the Emergence of a New Variant of Creutzfeldt-Jakob Disease.", Morbidity and Mortality Weekly Report, 12 April 1996: 295-303. http://www.cyber-dyne.com/~tom/Alzhe...d.html#and%20C * 'Currently four million Americans are diagnosed with Alzheimer's disease. The percentage of cases is on the rise with solid research showing that there are about 360,000 individuals newly diagnosed each year. At Yale University and the University of Pennsylvania at Pittsburgh, researchers recently studied the brains of people who died of Alzheimer's disease (46 in the Yale case and 54 in the Pittsburgh study). Surprisingly, the autopsies respectively showed that 13 percent and five percent of the dead were actually CJD cases misdiagnosed as Alzheimer's disease. In a third (smaller) study published in the Journal of Psychiatry and Neuroscience (1995), investigators reported that three out of 12 patients diagnosed with Alzheimer's disease were found to have had CJD when autopsied. It should be noted CJD symptoms may be remarkably similar to those of Alzheimer's disease. There are no accurate figures for the total number of U.S. Alzheimer's deaths each year, simply because it doesn't usually get reported as the cause of death. The Center for Disease Control (CDC) reported only 22,725 Alzheimer's deaths in 1998. However, a spokesperson for the National Alzheimer's Association, in a recent interview, agreed that the actual number could easily be 100,000, or even has high as 400,000 per year. Because Alzheimer's patients usually die within 8 to 10 years, she agreed that the CDC numbers must be grossly understated. If new research supports that approximately 10 percent of all Alzheimer's disease related deaths are in fact misdiagnosed CJD cases, then 10,000 to 40,000 CJD deaths will suddenly appear in America each year. That would be an epidemic. ' http://www.parkc.org/Madcow_veggies.html |
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"Torsten Brinch" > wrote in message ...
> We must be careful not to mix up things. There is a disease we can call > CJD (classical CJD), and another distinct from it, called vCJD (variant > CJD). Undoubtedly there have been people diagnosed with Alzheimer's > who really died from CJD, but not likely any from vCJD. vCJD affects > relatively young people. 'TSEs are a mysterious class of diseases that are called by different names in different species. For instance some identified types of TSE are Creutzfeldt-Jakob Disease (CJD), and its specific strain, new variant Creutzfeldt-Jakob Disease (nvCJD) which is a human disease apparently caused by the same agent which causes bovine spongiform encephalopathy (BSE) or British "mad cow" disease, Fatal Familial Insomnia (FFA), Gerstmann-Straussler-Scheinker Syndrome (GSS), scrapie in sheep, transmissible mink encephalopathy (TME) in mink in North America, and chronic wasting disease (CWD) in deer and elk in North America. There may be different strains of TSE within species, and new strains may be produced when TSEs move from one animal species to another. The common characteristics of TSE diseases are that they are invariably fatal. ' http://www.icta.org/legal/madcow.htm BSE prions propagate as either variant CJD-like or sporadic CJD-like prion strains in transgenic mice expressing human prion protein http://emboj.oupjournals.org/cgi/con...1/23/6358?etoc "Now people are beginning to realize that because something looks like sporadic CJD they can't necessarily conclude that it's not linked to (mad cow disease)," said Laura Manuelidis, section chief of surgery in the neuropathology department at Yale University, who conducted a 1989 study that found 13 percent of Alzheimer's patients actually had CJD. ...' http://www.nlm.nih.gov/medlineplus/n...ory_15312.html |
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![]() some useful facts on the UK BSE epidemic and mo http://www.defra.gov.uk/animalh/bse/...un02/order.pdf NB Watch out for linewrap. -- Oz This post is worth absolutely nothing and is probably fallacious. DEMON address no longer in use. |
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Oz > writes
>some useful facts on the UK BSE epidemic and mo [use monopitched font to view properly] ================ From: http://www.defra.gov.uk/animalh/bse/...un02/order.pdf UK cattle removed from the human food chain due BSE Year BSE cases OTMs ScS offspring Pre 1988 727 - 1988 2180 - - - 1989 7133 - - - 1990 14181 - - - 1991 25026 - - - 1992 36680 - - - 1993 34370 - - - 1994 23943 - - - 1995 14301 - - - 1996 8013 1156975 - - 1997 4309 848279 55465 - 1998 3179 891195 18956 1532 1999 2254 954766 1413 5844 2000 1311 963381 24 2559 2001 781 598601 0 1427 2002* 237 361257 0 1489 Total 178625 5774454** 75858 12851 OTMS = Over thirty months scheme ScS = Selective cull scheme offsping = offspring cull scheme ===================== SE Diagnoses in Exotic Species KUDU 6 GEMSBOK 1 NYALA 1 ORYX 2 ELAND 6 CHEETAH 5 PUMA 3 TIGER 3 OCELOT 3 BISON 1 ANKOLE COW 2 LION 4 Cheetah total excludes one in Australia and one in the Republic of Ireland (both of these were litter mates born in GB), and another two in France which were also born in GB. =================== SE Diagnoses in Domestic Cats Year Number of cases 1990 12 1991 12 1992 10 1993 11 1994 16 1995 8 1996 6 1997 6 1998 4 1999 2 2000 1 2001 1 ================= UK cases of all CJD's. [From: http://www.cjd.ed.ac.uk/figures.htm] Year Spor Iatro Fam GSS vCJD Total Deaths 1990 28 5 0 0 - 33 1991 32 1 3 0 - 36 1992 45 2 5 1 - 53 1993 37 4 3 2 - 46 1994 53 1 4 3 - 61 1995 35 4 2 3 3 47 1996 40 4 2 4 10 60 1997 60 6 4 1 10 81 1998 63 3 4 1 18 89 1999 62 6 2 0 15 85 2000 49 1 2 1 28 81 2001 57 3 3 2 20 85 2002 73 0 4 1 17 95 2003* 53 4 1 1 16 75 Total 687 44 39 20 137 927 Spor = Sporadic Iatro = Iatrogenic Fam = Familial GSS = Another inherited TE *To 1st dec 2003 -- Oz This post is worth absolutely nothing and is probably fallacious. DEMON address no longer in use. |
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On Mon, 5 Jan 2004 13:47:30 -0000, "pearl" > wrote:
>"Torsten Brinch" > wrote in message ... > >> We must be careful not to mix up things. There is a disease we can call >> CJD (classical CJD), and another distinct from it, called vCJD (variant >> CJD). Undoubtedly there have been people diagnosed with Alzheimer's >> who really died from CJD, but not likely any from vCJD. vCJD affects >> relatively young people. > >'TSEs are a mysterious class of diseases that are called by different >names in different species. For instance some identified types of TSE >are Creutzfeldt-Jakob Disease (CJD), and its specific strain, new >variant Creutzfeldt-Jakob Disease (nvCJD) which is a human disease >apparently caused by the same agent which causes bovine spongiform >encephalopathy (BSE) or British "mad cow" disease, Fatal Familial >Insomnia (FFA), Gerstmann-Straussler-Scheinker Syndrome (GSS), >scrapie in sheep, transmissible mink encephalopathy (TME) in mink in >North America, and chronic wasting disease (CWD) in deer and elk in >North America. There may be different strains of TSE within species, >and new strains may be produced when TSEs move from one animal >species to another. > >The common characteristics of TSE diseases are that they are >invariably fatal. ' >http://www.icta.org/legal/madcow.htm > >BSE prions propagate as either variant CJD-like or sporadic CJD-like >prion strains in transgenic mice expressing human prion protein >http://emboj.oupjournals.org/cgi/con...1/23/6358?etoc > >"Now people are beginning to realize that because something looks >like sporadic CJD they can't necessarily conclude that it's not linked >to (mad cow disease)," said Laura Manuelidis, section chief of surgery >in the neuropathology department at Yale University, who conducted >a 1989 study that found 13 percent of Alzheimer's patients actually >had CJD. ...' >http://www.nlm.nih.gov/medlineplus/n...ory_15312.html Again, we should be careful not to mix up things. It is one thing to say that some classical or sporadic CJD cases have been misdiagnosed as Alzheimer's (-that- cannot be doubted), another to say that exposure to BSE agent may cause both vCJD and classical CJD-like disease. Collinge's recent research (two references up from here) indicates that as perhaps more than a theoretical possibility. It will be awhile, though, probably decades, before there may or may not be actual epidemiological evidence that it is actually happening, from the ongoing UK CJD surveillance. There is a serious base-line problem, it would take more than just a small apparent increase in classical CJD prevalence to substantiate a link between BSE and classical CJD-like disease. |
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![]() "pearl" > wrote in message ... > "Jeff" > wrote in message ... > > > > "Rubystars" > wrote in message > > y.com... > > > > > > "Bill" > wrote in message > > > t... > > > > Is it still only one cow that has been found in the United States to have > > > > the disease? I mean, the article (what of it I read, it was very boring) > > > > made it sound as if the was some actual danger. Maybe thousands were > > > > found to be infected and I didn't read about it yet? > > > > > > There's been speculation that many of the people who were diagnosed with > > > Alzheimer's in the US may have actually had CJD. I thought I read something > > > about the brains of people who had died from Alzheimer's being studied and > > > the ones doing it finding out they had died of CJD, but I can't remember > > > where the article is now. > > > > > > -Rubystars > > > > > There is no proof what so ever that those inflicted with Alzheimer's had in > > fact CJD. There was one cow found to be diseased. They said millions would > > die in England when they had their scare and they had thousands of diseased > > cattle. They had a couple of deaths there. More liberal media hocus pocus. > > We had one cow in Canada and not one illness. These **** wads in the media > > don't care how they sell papers or get ratings. > > > > Jeff > > Alzheimer's And CJD Scientifically Linked > By Michael Greger > 12-29-03 > > (First published 6-16-96) > > If indeed a form of Bovine Spongiform Encephalopathy (BSE) > exists in the United States, one might expect to see a rise in the > number of cases of Creutzfeldt-Jakob disease (CJD). CJD, > however, is not a reportable illness in this country (Holman, 1995). > Because the Centers for Disease Control (CDC) does not actively > monitor the disease (Altman, 1996d) a rise similar to the one in > Britain could be missed (Altman, 1996d). Already, a number of > U.S. CJD clusters have been found. In the largest known U.S. > outbreak of sporadic cases to date(Flannery, 1996) a five-fold > expected rate was found to be associated with cheese > consumption in Pennsylvania's Lehigh Valley (Little, 1993) A > striking increase in CJD was also reported in Florida (Berger, > 1994) and there is an anecdotal report of an cluster in Oregon > (Boule, 1996). An analysis of death certificates in a number of > states, though, showed an overall stable and typical CJD > incidence rate from 1979 to 1993 (World, 1996). To track the > disease, the CDC has just initiated a four-state study of death > certificates (Altman, 1996a), but since it is considered well > known that death-certificate diagnoses are not always accurate > (Davanpour, 1993) the survey may not provide an accurate > assessment. > > The true prevalence of prion diseases in this or any other > country remains a mystery (Harrison, 1991). Compounding > the uncertainty, autopsies are rarely performed on atypical > dementias (Harrison, 1991), because medical professionals > fear infection (Altman, 1996a). The officially reported rate > in this country is less than 1 case in a million people per year > (World, 1996). An informal survey of neuropathologists, > however, registered a theoretical range of 2-12% of all > dementias as actually CJD (Harrison, 1991). And hundreds > of thousands of Americans suffer from severe dementias > every year (Brayne, 1994; United, 1995). Two other studies > average about a 3% CJD rate among dementia patients > (Mahendra, 1987; Wade, 1987). A preliminary 1989 > University of Pennsylvania study showed that 5% of patients > diagnosed with dementia were actually dying from > Creutzfeldt-Jakob disease (Boller, 1989). It would seem > CJD is seriously underdiagnosed at present (Harrison, 1991). > > The most common misdiagnosis of CJD is Alzheimer's > disease (Harrison, 1991). CJD was even described by > our government's top CJD researcher (Wlazelek, 1990a) > as "Alzheimer's in fast forward (Wlazelek, 1990b)." The > symptoms and pathology of both diseases overlap (Brown, > 1989). There can be spongy changes in Alzheimer's, for > example, and senile plaques in CJD (Brown, 1989). The > causes may overlap as well; epidemiological evidence > suggests that people eating meat more than four times a > week for a prolonged period have a three times higher > chance of suffering a dementia than long-time vegetarians > (Giem, 1993), although this result may be confounded by > vascular factors (Van Duijn, 1996). > > Paul Brown, medical director for the U.S. Public Health > Service (Gruzen, 1996), said that the brains of the young > people who died from the new CJD variant in Britain even > look like Alzheimer's brains (Hager, 1996). Stanley Prusinger, > the scientist who coined the term prion, speculates Alzheimer's > may in fact turn out to be a prion disease (Prusiner, 1984). In > younger victims the disease could look like multiple sclerosis > or a severe viral infection, according to Alzheimer's expert > Gareth Roberts (Brain, 1996). > > An estimated two to three million Americans are afflicted by > Alzheimer's (Scully, 1993); it is the fourth leading cause of > death among the elderly in the U.S (Perry, 1995). Twenty > percent or more of people clinically diagnosed with Alzheimer's > disease are found at autopsy to not have had Alzheimer's at all > (McKhann, 1984). At Yale, out of 46 patients clinically > diagnosed with Alzheimer's, 6 were proven to be CJD at > autopsy (Manuelidis, 1989). In another post-mortem study 3 > out of 12 "Alzheimer" patients actually died from a spongiform > encephalopathy (Teixeira, 1995). > > Carleton Gajdusek, who was awarded a Nobel Prize for his > work with prion diseases (Manuelidis, 1985), estimates that > 1% of people showing up in Alzheimer clinics actually have > CJD (Folstein, 1983). That means that hundreds* of people > (Hoyert, 1996; United, 1995) may already be dying from > mad cow disease each year in the United States. > > > WORKS CITED > > Altman, Lawrence K. "Four States Watching for Brain > Disorder." New York Times, 9 April 1996a. > > Altman, Lawrence K. "U.S. Officials Confident That Mad > Cow Disease of Britain Has Not Occured Here." New York > Times, 27 March 1996d: 12A. > > "Apocalypse Cow: U.S. Denials Deepen Mad Cow Danger." > PR Watch, 3.1 (1996): 1-8 > > Berger, Joseph R., et al. "Creutzfeldt-Jakob Disease: A > Ten-Year Experience." Neurology, 44 (1994): A260. > > Bleifuss, Joel. "Killer Beef." In These Times, 31 May 1993: 12-15. > > Boller, F., O. L. Lopez, and J. Moossy. "Diagnosis of Dementia. > " Neurology, 38 (1989): 76-79. > > Boule, Margie. "Despite Anectdotal Evidence, Docs Say No > Mad Cow Disease Here." Oregonian, 16 April 1996: C01. > > "Brain Disease May Be Commoner Than Thought - Expert." > Reuter Information Service, 15 May 1996. > > Brayne, C. "How Common are Cognitive Impairment and > Dementia?" Dementia and Normal Aging, Canbridge: University > Press, 1994. > > Brown, Paul. "Central Nervous System Amyloidoses." > Neurology, 39 (1989): 1103-1104. > > Davanpour, Zoreth, et al. "Rate of Creutzfeldt-Jakob Disease > in USA." Neurology, 43 (1993): A316. > > Flannery, Mary. "Twelve - Fifteen 'Mad Cow' Victims a Year > in Area." Philadelphia Daily News, 26 March 1996: 03. > > Folstein, M. "The Cognitive Pattern of Familial Alzheimer's > Disease." Biological Aspects of Alzheimer's Disease. Ed. R. > Katzman. Cold Spring Harbor Laboratory, 1983. > > Gruzen, Tara. "Sheep Parts Fail to Cause Mad Cow Disease > in U. S. Test." Seattle Times, 29 March 1996: A11. > > Hager, Mary and Mark Hosenball. "'Mad Cow Disease' in > the U.S.?" Newsweek, 8 April 1996:58-59. > > Harrison, Paul J., and Gareth W. Roberts. "'Life, Jim, But > Not as We Know It'? Transmissible Dementias and the Prion > Protein." British Journal of Psychiatry, 158 (1991): 457-70. > > Holman, R. C., et al. "Edidemiology of Creutzfeldt-Jakob > Disease in the United States, 1979-1990." Neuroepidemiology, > 14 (1995): 174-181. > > Hoyert, Donna L. "Vital and Health Statistics. Mortality > Trends for Alzheimer's Disease, 1979-1991." Washington: > U.S. Dept. of Health and Human Services, 1996. > > Little, Brian W., et al. "The Epidemiology of Creutzfeldt-Jakob > Disease in Eastern Pennsylvania." Neurology, 43 (1993): A316. > > Mahendra, B. Dementia, Lancaster: MTP Press Limited, > 1987: 174. > > Manuelidis, Elias E. "Presidential Address." Journal of > Neuropathology and Experimental Neurology, 44 (1985): 1-17. > > Manuelidis, Elias E. and Laura Manuelidis. "Suggested Links > between Different Types of Dementias: Creutzfeldt-Jakob > Disease, Alzheimer Disease, and Retroviral CNS Infections." > Alzheimer Disease and Associated Disorders, 2 (1989): 100-109. > > McKhann, Guy., et al. "Clinical Diagnosis of Alzheimer's Disease." > Neurology, 34 (1984): 939. > > Prusiner, S. "Some Speculations about Prions, Amyloid, and > Alzheimer's Disease." New England Journal of Medicine, 310 > (1984): 661-663. > > Perry, R.T., et al. "Human Prion Protein Gene: Two Different > 24 BP Deletions in an Atypical Alzheimer's Disease Family." > American Journal of Medical Genetics, 60 (1995): 12-18. > > Scully, R. E., et al. "Case Records of the Massachusetts > General Hospital." New England Journal of Medicine, 29 April > 1993: 1259-1263. > > Teixeira, F., et al. "Clinico-Pathological Correlation in Dementias." > Journal of Psychiatry and Neuroscience, 20 (1995): 276-282. > > United States Department of Commerce. Statistical Abstract > of the United States, Washington: Bureau of the Census, 1995. > > Van Duijn, C. M. "Epidemiology of the Dementia: Recent > Developments and New Approaches." Neuroepidemiology, > 60 (1996): 478-488. > > Van Duijn, C. M. "Epidemiology of the Dementia: Recent > Developments and New Approaches." Neuroepidemiology, > 60 (1996): 478-488. > > Wade, J. P. H., et al. "The Clinical Diagnosis of Alzheimer's > Disease." Archives of Neurology, 44 (1987): 24-29. > > Wlazelek, Ann. "Fatal Brain Disease Mystifies Experts." > Morning Call, 23 September 1990a: B01. > > Wlazelek, Ann. "Scientists Try to Track Fatal Disease; > International Expert Visits Area to Study Unusual Incedence > Rate." Morning Call, 27 September 1990b: B04. > > "World Health Organization Consultatoin on Public Health > Issues Related to Bovine Spongiform Encephalopathy and > the Emergence of a New Variant of Creutzfeldt-Jakob > Disease.", Morbidity and Mortality Weekly Report, 12 > April 1996: 295-303. > > http://www.cyber-dyne.com/~tom/Alzhe...d.html#and%20C > > * > 'Currently four million Americans are diagnosed with Alzheimer's disease. > The percentage of cases is on the rise with solid research showing that > there are about 360,000 individuals newly diagnosed each year. > > At Yale University and the University of Pennsylvania at Pittsburgh, > researchers recently studied the brains of people who died of Alzheimer's > disease (46 in the Yale case and 54 in the Pittsburgh study). Surprisingly, > the autopsies respectively showed that 13 percent and five percent of the > dead were actually CJD cases misdiagnosed as Alzheimer's disease. > > In a third (smaller) study published in the Journal of Psychiatry and > Neuroscience (1995), investigators reported that three out of 12 patients > diagnosed with Alzheimer's disease were found to have had CJD when > autopsied. It should be noted CJD symptoms may be remarkably similar > to those of Alzheimer's disease. > > There are no accurate figures for the total number of U.S. Alzheimer's > deaths each year, simply because it doesn't usually get reported as the > cause of death. The Center for Disease Control (CDC) reported only > 22,725 Alzheimer's deaths in 1998. However, a spokesperson for the > National Alzheimer's Association, in a recent interview, agreed that the > actual number could easily be 100,000, or even has high as 400,000 > per year. Because Alzheimer's patients usually die within 8 to 10 years, > she agreed that the CDC numbers must be grossly understated. If new > research supports that approximately 10 percent of all Alzheimer's > disease related deaths are in fact misdiagnosed CJD cases, then 10,000 > to 40,000 CJD deaths will suddenly appear in America each year. > That would be an epidemic. ' > http://www.parkc.org/Madcow_veggies.html > > > > > > > |
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![]() "Jim Webster" > wrote in message ... > > "Torsten Brinch" > wrote in message > ... > > On Mon, 5 Jan 2004 03:00:20 -0600, "Jeff" > wrote: > > > > > > > >"Rubystars" > wrote in message > > gy.com... > > > > >> There's been speculation that many of the people who were diagnosed > with > > >> Alzheimer's in the US may have actually had CJD. I thought I read > > >>something > > >> about the brains of people who had died from Alzheimer's being studied > and > > >> the ones doing it finding out they had died of CJD, but I can't > remember > > >> where the article is now. > > > > >There is no proof what so ever that those inflicted with Alzheimer's had > in > > >fact CJD. > > > > We must be careful not to mix up things. There is a disease we can call > > CJD (classical CJD), and another distinct from it, called vCJD (variant > > CJD). Undoubtedly there have been people diagnosed with Alzheimer's > > who really died from CJD, but not likely any from vCJD. vCJD affects > > relatively young people. > > > > >There was one cow found to be diseased. They said millions would > > >die in England when they had their scare and they had thousands of > diseased > > >cattle. <snip> > > > > Pointedly one could say some said there was only a relatively few BSE > > clinical cases at one stage during the UK epizootic , with the case > > number appearing to be leveling out at a low prevalence. Downplaying > > it, so maybe it was no big deal, one could wait and see. However, they > > were sitting on a huge number of subclinical cases, the proverbial > > hidden part of the ice-berg they just could not see yet, and we are to > > be happy that some were sufficiently foresighted to act accordingly. > > note that when people have their tonsils out, these are now, in many parts > of the UK if not all of it, tested for nvCJD.. The idea being that these > tissues are among the first parts to become infective and therefore will be > a useful marker as to the number of cases. So far none have come up >infected If they can't do anything to cure the person or prevent onset of the disease, then what's the point? -Rubystars |
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On Mon, 05 Jan 2004 18:08:23 GMT, "Rubystars" >
wrote: >"Jim Webster" > wrote in message ... >> note that when people have their tonsils out, these are now, in many parts >> of the UK if not all of it, tested for nvCJD.. The idea being that these >> tissues are among the first parts to become infective and therefore will >> be a useful marker as to the number of cases. So far none have come up >>infected > >If they can't do anything to cure the person or prevent onset of the >disease, then what's the point? Rubystars, don't take anything from that maroon. You can throw him a stick, and he'll get the wrong end 8 out of ten. Even an oyster can do better. |
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On Mon, 05 Jan 2004 19:42:42 +0100, Torsten Brinch
> wrote: >On Mon, 05 Jan 2004 18:08:23 GMT, "Rubystars" > >wrote: >>"Jim Webster" > wrote in message ... > >>> note that when people have their tonsils out, these are now, in many parts >>> of the UK if not all of it, tested for nvCJD.. The idea being that these >>> tissues are among the first parts to become infective and therefore will >>> be a useful marker as to the number of cases. So far none have come up >>>infected >> >>If they can't do anything to cure the person or prevent onset of the >>disease, then what's the point? > >Rubystars, don't take anything from that maroon. You can throw him >a stick, and he'll get the wrong end 8 out of ten. Even an oyster can do >better. Except, of course, that Mr. Webster was correct in his posting. What kind of an oyster do you think he is? Perhaps neither you nor Ms Ruby knows much about epidemiology, CJD or nvCJD. Boron |
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On Mon, 05 Jan 2004 13:54:57 -0500, Boron Elgar
> wrote: >On Mon, 05 Jan 2004 19:42:42 +0100, Torsten Brinch > wrote: > >>On Mon, 05 Jan 2004 18:08:23 GMT, "Rubystars" > >>wrote: >>>"Jim Webster" > wrote in message ... >> >>>> note that when people have their tonsils out, these are now, in many parts >>>> of the UK if not all of it, tested for nvCJD.. The idea being that these >>>> tissues are among the first parts to become infective and therefore will >>>> be a useful marker as to the number of cases. So far none have come up >>>>infected >>> >>>If they can't do anything to cure the person or prevent onset of the >>>disease, then what's the point? >> >>Rubystars, don't take anything from that maroon. You can throw him >>a stick, and he'll get the wrong end 8 out of ten. Even an oyster can do >>better. > >Except, of course, that Mr. Webster was correct in his posting. ><snip> Come, show me what you've got to support that. :-) |
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![]() Oz's big brother > wrote in message ... > > "Paul M. Cook©®" > wrote in message > ... > > > > So you're saying that if prions could vote, they'd vote democrat? > > As Oz is a Brit, this comment is as meaningless as those I have snipped > > I suggest you actually check a few facts What I'll do is check who I reply to next time. I was quotiong the person he was quoting. Shouldn;t post so early in the AM. Paul |
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![]() "Oz" > wrote in message ... > Paul M. Cook©® > writes > > > >So you're saying that if prions could vote, they'd vote democrat? > > Eh? Posted that to the wrong person. Should have been for the one you replied to. Paul > >This > >reminds me so much of the imbeciles that refuted the devastating affects of > >DDT back in the 60's. They were the one ultimately proven wrong, now > >weren't they? One stalwart individual even sprinkled it on his breakfast > >cereal to prove it was all just "hocus pocus." He died, at a very young > >age, of liver cancer a few years later. > > BSE and prions are not fresh news to the UK. > Take a look at the date of the first case. > > >Basic husbandry practices in Europe dating back 8 centuries dictated that > >animals not be fed the remains of their own species. > > Pigs have been fed pigswill containing pork for much longer than that. > Chickens will happily cannibalise each other. > So you are patently wrong. > That's even before we get into the well documented cases of ruminants > knawing ones and eating eggs and chicks. > > >Seems a few lessons > >have been forgotten. > > Seems you are a tad short of actually knowing what you are talking > about. Well not entirely, no. Basic animal husbandry practices for centuries, including the ME and Europe did not include cannibalizing animals. Now if some of them did so on their own, not much you could do to stop it. A chicken eating another chicken is not husbandry practices. > >But by all means label every scientist out to > >enlighten and protect the public some wild-eyed radical. > > Hardly. > > >Have some DDT with your Cheerios and think about it. > > Another effort at emotive nonsense. Again, I was posting that to the personj you responded to. And it's an example of the same mental;ity we're seeing again. This whole thing will be dismissed as nonsense until it reaches an epidemic. That was my point. Paul |
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Torsten Brinch > writes
>Come, show me what you've got to support that. :-) 20 April 2000 A Scientific Committee chaired by Professor Borysiewicz of University of Wales College of Medicine met to review analyses taken from 3,000 specimens of human tonsil and appendix tissue. -- Oz This post is worth absolutely nothing and is probably fallacious. DEMON address no longer in use. |
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Paul M. Cook©® > writes
>> Pigs have been fed pigswill containing pork for much longer than that. >> Chickens will happily cannibalise each other. >> So you are patently wrong. >> That's even before we get into the well documented cases of ruminants >> knawing ones and eating eggs and chicks. >> >> >Seems a few lessons >> >have been forgotten. >> >> Seems you are a tad short of actually knowing what you are talking >> about. > >Well not entirely, no. Basic animal husbandry practices for centuries, >including the ME and Europe did not include cannibalizing animals. See pigs and swill, above .... Or do you think people with household pigsty's didn't actually eat their pigs? >Now if >some of them did so on their own, not much you could do to stop it. A >chicken eating another chicken is not husbandry practices. I suspect the chicken rearers would disagree. MBM/bonemeal etc has been fed to cattle for a very long time. -- Oz This post is worth absolutely nothing and is probably fallacious. DEMON address no longer in use. |
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![]() "Oz" > wrote in message ... > Paul M. Cook©® > writes > >> Pigs have been fed pigswill containing pork for much longer than that. > >> Chickens will happily cannibalise each other. > >> So you are patently wrong. > >> That's even before we get into the well documented cases of ruminants > >> knawing ones and eating eggs and chicks. > >> > >> >Seems a few lessons > >> >have been forgotten. > >> > >> Seems you are a tad short of actually knowing what you are talking > >> about. > > > >Well not entirely, no. Basic animal husbandry practices for centuries, > >including the ME and Europe did not include cannibalizing animals. > > See pigs and swill, above .... > Or do you think people with household pigsty's didn't actually eat their > pigs? Boy you're a stubborn knob. I said husbandry practices and you imply some sort of international law. I suppose some of the farmers bred animals from the same parents too. What does that prove? Some were too stupid to follow the wisdom of the day? > >Now if > >some of them did so on their own, not much you could do to stop it. A > >chicken eating another chicken is not husbandry practices. > > I suspect the chicken rearers would disagree. Well chickens don't read, that's true. Paul |
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![]() "Paul M. Cook©®" > wrote in message ... > > Oz's big brother > wrote in message > ... > > > > "Paul M. Cook©®" > wrote in message > > ... > > > > > > So you're saying that if prions could vote, they'd vote democrat? > > > > As Oz is a Brit, this comment is as meaningless as those I have snipped > > > > I suggest you actually check a few facts > > What I'll do is check who I reply to next time. I was quotiong the person > he was quoting. Shouldn;t post so early in the AM. > A lot of the problem is the cross posts. Oz is posting on sci.agriculture. This, I suspect, may be a group with a wider international focus that alt.politics.bush? :-)) Jim Webster > Paul > > > |
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"Rubystars" > wrote in message
. com... >> > note that when people have their tonsils out, these are now, in many parts > > of the UK if not all of it, tested for nvCJD.. The idea being that these > > tissues are among the first parts to become infective and therefore will > be > > a useful marker as to the number of cases. So far none have come up > >infected > > If they can't do anything to cure the person or prevent onset of the > disease, then what's the point? it is quite simple. We are monitoring a population, which is rather bigger and probably more important than a person. If the disease is not present in a population, then the person cannot get it. If the disease is present in a population, then it is a risk to the individuals. Also remember that there have been claims that millions of UK citizens will die. When we callously refused to die and thus put a lot of lobby groups noses out of joint. So they said actually we were just incubating it and would all die later (which is in a way true, everyone dies later) so they run these tests on tonsils and discover that in our brutally uncooperative way, we aren't even incubating it. Jim Webster |
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Paul M. Cook©® > writes
>> >Well not entirely, no. Basic animal husbandry practices for centuries, >> >including the ME and Europe did not include cannibalizing animals. >> >> See pigs and swill, above .... >> Or do you think people with household pigsty's didn't actually eat their >> pigs? > >Boy you're a stubborn knob. I said husbandry practices and you imply some >sort of international law. I suppose some of the farmers bred animals from >the same parents too. What does that prove? Some were too stupid to follow >the wisdom of the day? Outside rather recent times most of the pigs would have been housepigs fed on swill. That was the husbandry practice of the times, everyone would have done it. >> >Now if >> >some of them did so on their own, not much you could do to stop it. A >> >chicken eating another chicken is not husbandry practices. >> >> I suspect the chicken rearers would disagree. > >Well chickens don't read, that's true. Nor would most of the farmers in days past. Or most of the population, come to that. -- Oz This post is worth absolutely nothing and is probably fallacious. DEMON address no longer in use. |
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"Jim Webster" > wrote in message ...
> "Rubystars" > wrote in message > . com... > >> > note that when people have their tonsils out, these are now, in many > parts > > > of the UK if not all of it, tested for nvCJD.. The idea being that these > > > tissues are among the first parts to become infective and therefore will > > be > > > a useful marker as to the number of cases. So far none have come up > > >infected > > > > If they can't do anything to cure the person or prevent onset of the > > disease, then what's the point? > it is quite simple. We are monitoring a population, which is rather bigger > and probably more important than a person. If the disease is not present in > a population, then the person cannot get it. If the disease is present in a > population, then it is a risk to the individuals. > Also remember that there have been claims that millions of UK citizens will > die. When we callously refused to die and thus put a lot of lobby groups > noses out of joint. So they said actually we were just incubating it and > would all die later (which is in a way true, everyone dies later) so they > run these tests on tonsils and discover that in our brutally uncooperative > way, we aren't even incubating it. > > Jim Webster '..the director of the UK CJD Surveillance Unit warned that current tests might vastly under-represent the risk of infection. Professor James Ironside told the BBC last night that the tests were not sensitive enough to identify all those infected with the human form of bovine spongiform encephalopathy (BSE). He said: "Because of the nature of the specimens used in the study we have had to employ a technique that is not as sensitive as other methods that are currently available so in some ways even this finding might be an under-representation of the actuality in terms of infection with BSE." http://www.guardian.co.uk/bse/articl...795882,00.html |
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![]() "pearl" > wrote in message ... > > it is quite simple. We are monitoring a population, which is rather bigger > > and probably more important than a person. If the disease is not present in > > a population, then the person cannot get it. If the disease is present in a > > population, then it is a risk to the individuals. > > Also remember that there have been claims that millions of UK citizens will > > die. When we callously refused to die and thus put a lot of lobby groups > > noses out of joint. So they said actually we were just incubating it and > > would all die later (which is in a way true, everyone dies later) so they > > run these tests on tonsils and discover that in our brutally uncooperative > > way, we aren't even incubating it. > > > > Jim Webster > > '..the director of the UK CJD Surveillance Unit warned that current > tests might vastly under-represent the risk of infection. > > Professor James Ironside told the BBC last night that the tests were > not sensitive enough to identify all those infected with the human form > of bovine spongiform encephalopathy (BSE). > So what. We have found NONE! Sorry and all that, but there will be no huge corpse fest. 3000 tissue samples. So if we were going to get 4 million dead (a figure much mooted by those shroud waving to get their grant allocation increased in the early days) then we would expect out of a population of 60 million (round figures) we would expect one in 15 samples to be infected Out of 3000 samples that would be 200. So the test isn't that sensitive, we might miss 50%. Fine, we would expect 100 positive samples. Yet there are NONE The Guardian has papers to sell and James Ironside obviously needs more funding for surveillance, after all he doesn't want the UK CJD Surveillance Unit to be wound down because there is nothing out there to look for. Jim Webster |
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![]() "Jim Webster" > wrote in message ... > "Rubystars" > wrote in message > . com... > >> > note that when people have their tonsils out, these are now, in many > parts > > > of the UK if not all of it, tested for nvCJD.. The idea being that these > > > tissues are among the first parts to become infective and therefore will > > be > > > a useful marker as to the number of cases. So far none have come up > > >infected > > > > If they can't do anything to cure the person or prevent onset of the > > disease, then what's the point? > it is quite simple. We are monitoring a population, which is rather bigger > and probably more important than a person. If the disease is not present in > a population, then the person cannot get it. If the disease is present in a > population, then it is a risk to the individuals. > Also remember that there have been claims that millions of UK citizens will > die. When we callously refused to die and thus put a lot of lobby groups > noses out of joint. So they said actually we were just incubating it and > would all die later (which is in a way true, everyone dies later) so they > run these tests on tonsils and discover that in our brutally uncooperative > way, we aren't even incubating it. > > Jim Webster I'm glad that it hasn't been found in the tonsils yet. I hope there aren't a lot of people in the US who are incubating it. -Rubystars |
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On Mon, 5 Jan 2004 21:34:46 -0000, "Jim Webster"
> wrote: > >"pearl" > wrote in message ... >> > it is quite simple. We are monitoring a population, which is rather >bigger >> > and probably more important than a person. If the disease is not present >in >> > a population, then the person cannot get it. If the disease is present >in a >> > population, then it is a risk to the individuals. >> > Also remember that there have been claims that millions of UK citizens >will >> > die. When we callously refused to die and thus put a lot of lobby groups >> > noses out of joint. So they said actually we were just incubating it and >> > would all die later (which is in a way true, everyone dies later) so >they >> > run these tests on tonsils and discover that in our brutally >uncooperative >> > way, we aren't even incubating it. >> > >> > Jim Webster >> >> '..the director of the UK CJD Surveillance Unit warned that current >> tests might vastly under-represent the risk of infection. >> >> Professor James Ironside told the BBC last night that the tests were >> not sensitive enough to identify all those infected with the human form >> of bovine spongiform encephalopathy (BSE). >> > >So what. We have found NONE! Sorry and all that, but there will be no huge >corpse fest. > >3000 tissue samples. >So if we were going to get 4 million dead (a figure much mooted by those >shroud waving to get their grant allocation increased in the early days) >then we would expect out of a population of 60 million (round figures) we >would expect one in 15 samples to be infected > >Out of 3000 samples that would be 200. > > >So the test isn't that sensitive, we might miss 50%. Fine, we would expect >100 positive samples. Yet there are NONE >The Guardian has papers to sell and James Ironside obviously needs more >funding for surveillance, after all he doesn't want the UK CJD >Surveillance Unit to be wound down because there is nothing out there to >look for. > >Jim Webster Nothing worse then a deaf, dumb and blind country bumpkin farmer who earns a living cutting corners. This is the same arsehole who thinks F&M, Pesticide and herbicide danger, factory farming abuses etc are all a figment of our imagination. Your logic makes about as much sense as your ideas on good farming practice, not to mention your ability to claim you own land you don't!!! ********************************************** 'You can't win 'em all.' Lord Haw Haw. Since I stopped donating money to CONservation hooligan charities Like the RSPB, Woodland Trust and all the other fat cat charities I am in the top 0.801% richest people in the world. There are 5,951,930,035 people poorer than me If you're really interested I am the 48,069,965 richest person in the world. And I'm keeping the bloody lot. So sue me. http://www.globalrichlist.com/ Newsgroup ettiquette 1) Tell everyone the Trolls don't bother you. 2) Say you've killfiled them, yet continue to respond. 3) Tell other people off who repsond despite doing so yourself. 4) Continually talk about Trolls while maintaining they're having no effect. 5) Publicly post killfile rules so the Trolls know how to avoid them. 6) Make lame legal threats and other barrel scraping manoeuvres when your abuse reports are ignored. 7) Eat vast quantities of pies. 8) Forget to brush your teeth for several decades. 9) Help a demon.local poster with their email while secretly reading it. 10) Pretend you're a hard ******* when in fact you're as bent as a roundabout. 11) Become the laughing stock of Usenet like Mabbet 12) Die of old age 13) Keep paying Dr Chartham his fees and hope one day you will have a penis the girls can see. --------------------------------------- "If you would'nt talk to them in a bar, don't *uckin' vote for them" "Australia was not *discovered* it was invaded" The Big Yin. |
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Rubystars > writes
>I'm glad that it hasn't been found in the tonsils yet. I hope there aren't a >lot of people in the US who are incubating it. Ye gods.... One case in an imported canadian cow and they think they are at risk.... -- Oz This post is worth absolutely nothing and is probably fallacious. DEMON address no longer in use. |
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pearl > writes
>Professor James Ironside told the BBC last night that the tests were >not sensitive enough to identify all those infected with the human form >of bovine spongiform encephalopathy (BSE). Big deal. If it's another 100 over 10 years or another 120, it's a very very rare way to die. >He said: "Because of the nature of the specimens used in the study >we have had to employ a technique that is not as sensitive as other >methods that are currently available so in some ways even this finding >might be an under-representation of the actuality in terms of infection >with BSE." And of course he has no research institute to protect from funding cuts ..... -- Oz This post is worth absolutely nothing and is probably fallacious. DEMON address no longer in use. |
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